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- Create Date July 25, 2017
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LL was a term male infant transferred from an outlying hospital with respiratory distress and apnea. Mother was G1, P0 and GBS negative. She was admitted for induction at 39½ weeks gestation after an uneventful pregnancy until pregnancy-induced hypertension appeared a few days prior to delivery. Membranes were ruptured 28 hours prior to delivery; fluid was clear. She developed a fever of 100.8 degrees during labor, while receiving epidural analgesia. There was no evidence of fetal distress. A single dose of Ancef was given, then C-section was performed for failure
to progress.
Apgar scores were 8 and 8; birth weight was 3030 grams, with symmetrical OFC and length measurements. The baby required supplemental O2 in the delivery room, then continued to have mild grunting and tachypnea in the nursery. He was placed in hood O2, and stabilized in 36% O2. A blood culture and CBC wereobtained, and he was started
Please download following .PDF to view the full clinical case study.
Robert White, MD
Director, Regional Newborn Program, Memorial Hospital
South Bend, IN, USA
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Upcoming Events
December
Event Details
Q4 2025
Event Details
Q4 2025
Time
October 1, 2025 - December 31, 2025 (All Day)(GMT+08:00)
Location
China
Register NOW
Upcoming LIVE eSeminars
December
18dec11:00 am12:00 pmHow to Interpret SEEG RecordingsLive eSeminar
Event Details
SEEG interpretation relies on the compartmentalization of the epileptogenic region into ‘lesional’, ‘irritative’, and ‘epileptogenic’ zones to differentiate interictal and ictal abnormalities. This approach avoids any bias in localization, i.e.
Event Details
SEEG interpretation relies on the compartmentalization of the epileptogenic region into ‘lesional’, ‘irritative’, and ‘epileptogenic’ zones to differentiate interictal and ictal abnormalities. This approach avoids any bias in localization, i.e. causing confusion between lesion and epilepsy, or between spikes and seizures. We will review what these different terms mean, which are the relevant biomarkers that help to differentiate these 3 zones, what are their causal relationship, and how their identification then allows, through their 3D representation, a careful individualized planning of the surgical resection.
Learning Objectives:
- Understand that SEEG interpretation starts with electrode implantation
- Identify relevant paroxysmal activities
- Identify relevant seizure onset patterns
- Understand how to use stimulation
1.0 CEU is available through ASET
Speakers for this event
-
Prof. Philippe Kahane
Prof. Philippe Kahane
Philippe Kahane, MD, PhD, is a neurologist and neurophysiologist, Hospital Practitioner and University Professor at Grenoble-Alpes Hospital & University, France. He is heading the Clinical Neurosciences Axis of Grenoble Hospital. He is acknowledged as an international expert on presurgical assessment of drug-resistant epilepsies in adults and children, including SEEG recordings. The area of research covers various fields in epileptology and physiology in humans, including the characterization of epileptogenic networks using SEEG recordings and stimulation, the assessment of physiological networks by analysing SEEG oscillatory responses to different cognitive tasks, and the implementation of novel surgical therapies such as deep brain stimulation. He is Associate Editor of the international journal Epileptic Disorders, and is co-director of the annual Asian, European and North-American SEEG Training Courses. He is author or co-author of over 280 articles in international journals indexed in Medline, and has been invited to give presentations in over 320 international seminars and meetings.
Time
December 18, 2025 11:00 am - 12:00 pm(GMT-05:00)
